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Effexor (Venlafaxine)

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Generic Effexor is a qualitative medication which is taken in treatment of panic disorder, anxiety and depression. Generic Effexor effectiveness is in balancing the brain. It is a SSNRIs (selective serotonin and norepinephrine reuptake inhibitors).

Other names for this medication:
Agrofan, Alventa, Anapresin, Argofan, Axyven, Benolaxe, Depant prolong, Deprevix, Deprexor, Depurol, Desinax, Dobupal, Efaxil, Efaxin, Efectin, Efectin er, Efetrin, Efevelone, Efexiva, Efexor, Efexor exel, Effexor, Elafax, Elify, Faxine, Faxiprol, Flavix, Ganavax, Idoxen, Ireven, Jarvis, Lafax, Lanvexin, Laroxin, Melocin, Memomax, Mezine, Mollome, Nervix, Nopekar, Norafexine, Norpilen, Odven, Olwexya, Prefaxine, Quilarex, Ranfaxiran, Senexon, Sentidol, Sesaren, Subelan, Tavex, Tifaxin, Trevilor, Valax, Valosine, Vandral, Vedixal, Velafax, Velaxin, Venax, Venaxibene, Venex, Venexor, Veniz, Venla, Venlaf, Venlafab, Venlafaxina, Venlafaxinum, Venlagamma, Venlalek, Venlalic, Venlasan, Venlax, Venlax er, Venlaxor, Venlectine, Venlift, Venlix, Venlofex, Vennaxa, Vensir, Viepax, Voxatin

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Also known as:  Venlafaxine.


Generic Effexor is a perfect remedy in struggle against panic disorder, anxiety. Its target is to treat depression. Generic Effexor effectiveness is in balancing the brain. It is a SSNRIs (selective serotonin and norepinephrine reuptake inhibitors).

Generic name of Generic Effexor is Venlafaxine.

Effexor is also known as Venlafaxine, Ventab, Efexor, Venlor, Venla, Venlift.

Brand names of Generic Effexor are Effexor, Effexor XR.


Generic Effexor is available in tablets and capsules. Generic Effexor is taken orally with food.

Do not crush or chew it.

Take Generic Effexor at the same time every day with water.

If you want to achieve most effective results do not stop taking Generic Effexor suddenly.


If you overdose Generic Effexor and you don't feel good you should visit your doctor or health care provider immediately.


Store at a controlled temperature between 20 and 25 degrees C (68 degrees and 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Effexor are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Generic Effexor if you are allergic to Generic Effexor components.

Do not take Generic Effexor if you are pregnant, planning to become pregnant, or are breast-feeding.

Do not take it if you are under 18.

Be careful with Generic Effexor if you take ketoconazole (such as Nizoral); other antidepressants (fluoxetine (such as Prozac), sertraline (such as Zoloft), amoxapine (such as Ascendin), paroxetine (such as Paxil), citalopram (such as Celexa), protriptyline (such as Vivactil), clomipramine (such as Anafranil), trimipramine (such as Surmontil), desipramine (such as Norpramin), escitalopram (such as Lexapro), fluvoxamine (such as Luvox), imipramine (such as Tofranil), amitriptyline (such as Elavil), nortriptyline (such as Pamelor)); imetidine ( such as Tagamet HB, Tagamet); tryptophan; zolmitriptan (such as Zomig); rizatriptan (such as Maxalt), almotriptan (such as Axert), frovatriptan (such as Frova), naratriptan (such as Amerge), sumatriptan (such as Imitrex); warfarin (such as Coumadin); risperidone (such as Risperdal) or haloperidol (such as Haldol), monoamine oxidase inhibitors (MAOIs).

Avoid alcohol.

Be careful when you are driving or operating machinery.

It can be dangerous to stop Generic Effexor taking suddenly.

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One hundred patients with GAD, diagnosed according to Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition-Text Revision criteria, were enrolled and their main demographic and clinical features collected. Patients were then treated with selective serotonin reuptake inhibitors or venlafaxine for 8 weeks in open-label conditions. Treatment response and other clinical variables were analyzed after dividing the sample into two groups according to DUI (DUI 12 months).

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The effects of long-term administrations of a low (10 mg/kg/day) and a high (40 mg/kg/day) dose of the dual 5-HT and NE reuptake inhibitor venlafaxine (delivered s.c. by osmotic minipumps for 21 days) were assessed on the electrically-evoked release of tritium from hippocampal slices preloaded with either [(3)H]5-HT or [(3)H]NE, 48 h after the removal of the minipump. The high, but not the low, dose regimen of venlafaxine enhanced the electrically-evoked release of [(3)H]5-HT while treatment with the high dose of venlafaxine failed to alter the electrically-evoked release of [(3)H]NE. The inhibitory effect of the 5-HT(1B) agonist CP 93,129 on the electrically evoked release of [(3)H]5-HT was unaltered by the low dose regimen of venlafaxine while it was attenuated in rats treated with the high dose of venlafaxine, indicative of a functional desensitization of the terminal 5-HT(1B) autoreceptor. Unexpectedly, neither regimen of venlafaxine altered the inhibitory effect of UK 14,304 on the electrically evoked release of both [(3)H]5-HT and [(3)H]NE, indicating that neither the alpha(2)-adrenergic auto- nor heteroreceptors were desensitized. Finally, the functions of the 5-HT and NE reuptake process were assessed. None of the treatment regimens altered the basal uptake of [(3)H]5-HT from hippocampal or mesencephalic slices nor that of [(3)H]NE from hippocampal slices. Finally, the enhancing effect of 1 microM of paroxetine in the perfusion medium on the electrical release of [(3)H]5-HT was unaltered in hippocampal slices prepared from rats that had been treated for 21 days with 40 mg/kg/day of venlafaxine. Taken together, these results indicate that, in terms of alteration of the sensitivity of the terminal 5-HT(1B) autoreceptor, alpha(2)-adrenergic auto-and heteroreceptors, the effects of long-term administration of venlafaxine are no different than those observed with classical SSRI's.

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Data were obtained from 9 individual health plans with more than 1.1 million covered lives affiliated with a national managed-care organisation.

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Choosing an antidepressant for a medically ill patient is a common clinical problem. There is an increased prevalence of depression among patients with many medical illnesses. The clinician must be familiar with the recent additions to the antidepressant armamentarium, including the synaptic pharmacology and pharmacokinetics of the available agents. The factors usually used to guide antidepressant selection must be augmented by a careful evaluation of the patient's medical illness and therapeutic regimen. Special attention must be paid to the potential for drug-illness and drug-drug interactions. The author has used the example of four illnesses commonly associated with depression--stroke, cancer, Parkinson's disease, and dementia--to illustrate how these considerations affect the choice of an antidepressant.

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The reduction of prefrontal theta QEEG cordance value after first week of treatment might be helpful in the prediction of response to venlafaxine.

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Metabonomic approach is helpful to further understanding the pathophysiology of depression and assisting in clinical diagnosis of depression and is also a valuable tool for studying the essence of Chinese medicine's syndrome theory and therapeutic effect mechanism of TCM.

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To evaluate the efficacy, safety, and tolerability of flexible-dose venlafaxine ER compared with placebo in the short-term treatment of generalized social anxiety disorder and, secondarily, to compare paroxetine with venlafaxine ER and paroxetine with placebo.

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We report a case of hyponatremia probably caused by venlafaxine. Awareness of this potential problem would be helpful to clinicians and should be considered in the differential diagnosis of hyponatremia.

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Hepatic injury has been reported following duloxetine use. This study further examines the hepatic safety of duloxetine in a large US health insurance database.

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A CT scan documented signs of cortical atrophy in our patient's brain but excluded vascular brain injury, while clinical evaluation and anamnesis excluded a relationship between hallucinations and cortical atrophy. Genetic and pharmacologic factors may be involved in venlafaxine-induced adverse effects. Venlafaxine is metabolized primarily by CYP2D6 and is a substrate of P-glycoprotein. Propafenone, a known substrate and inhibitor of both CYP2D6 and P-glycoprotein, could therefore be involved in venlafaxine-induced hallucinations through the increase of venlafaxine plasma concentrations.

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These results, together with those of previously reported studies, suggest that venlafaxine has antidepressant efficacy comparable to that provided by available antidepressants.

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effexor replacement drug 2016-11-03

Fifty male adult SD rats were randomly divided into control group, model group and Singulair Pills three treatment groups (5,10, 20 mg*kg(-1) venlafaxine) with ten in each group. After the procedure of selective cerebral right middle artery embolism, a paradigm of continuous 3-week chronic unpredictable mild stress (CUMS) was used to induce PSD. Along with the course of CUMS the peritoneal injection at different dose levels of venlafaxine were performed once a day in PSD rats in a fixed time interval. Morris water maze test was applied to assess the spatial learning and memory function and immunohistochemical staining was used to detect the change of BDNF expression.

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Conditioned stimulus properties of venlafaxine are still unknown. In the present study, the discriminative stimulus properties of venlafaxine by using a conditioned taste aversion procedure were investigated. Swiss Webster mice were allowed to reach water from 2 pipettes for 20 min (09:00-11:30 h), plus 30 min (15:30-16:00 h), daily. During the 4 days, the test drugs [fluoxetine, escitalopram, tianeptine, reboxetine, and Nomega-nitro-L-arginine methyl ester (L-NAME)] were injected to mice at least 1 h after they had first water session. On day 5, they consumed glucose solution (5% w/v) and immediately injected with conditioning drug (venlafaxine 32 mg/kg). On day 8, mice were allowed to make a choice between water and glucose solution. The amount of glucose consumption as a percentage of total fluid intakes was calculated for each animal. Significant reduction in glucose choice was defined as conditioned taste aversion. Venlafaxine (32 mg/kg) induced a robust conditioned taste aversion in mice. Pre-exposure to tianeptine (2.5-10 mg/kg), fluoxetine (10 mg/kg), escitalopram (32 mg/kg), and reboxetine (5 mg/kg) substituted for venlafaxine by preventing the conditioned taste aversion induced by venlafaxine. L-NAME did not substitute for venlafaxine. Substitution of venlafaxine by fluoxetine, tianeptine, escitalopram, and reboxetine provides further evidence that both Buspar 2 Mg 5-HT and noradrenaline reuptake inhibition may play an important role in the stimulus effect of venlafaxine.

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In this Zocor With Alcohol study, switching to the weak CYP2D6 inhibitor escitalopram was safe and feasible and resulted in clinically relevant rises in endoxifen concentrations. We therefore advise switching paroxetine and fluoxetine to escitalopram in patients using tamoxifen. However, switching should always be weighed in individual patients.

effexor medicine 2016-10-16

161 patients with major depressive disorder were included in an open-label, multicentre clinical study. All patients were treated with venlafaxine extended release in flexible doses ranging from 75 to 325 mg daily over an 8-week period. Efficacy Amaryl Dosage Form measurements included the 17-item Hamilton Depression Scale, the Clinical Global Impression-Improvement scale (CGI-I), the Severity of Illness scale (CGI-S), and the Depression and Somatic Symptom Scale (DSSS). All scales were administered at baseline and at weeks 2, 4 and 8.

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Outcomes from published, randomized, placebo-controlled trials reporting on moderately Nizoral Generic to severely depressed patients (Hamilton Rating Scale for Depression [HAM-D] > or =15 or Montgomery-Asberg Depression Rating Scale [MADRS] > or =18). A systematic literature search of Cochrane, EMBASE, and MEDLINE (1996-January 2005) was performed. Two independent reviewers judged the trials for acceptance, and last observation carried forward data were extracted. Differences in remission (8-week HAM-D score < or =7 or MADRS < or =10), response (50% decrease on either scale), and dropout rates from lack of efficacy and adverse events were meta-analyzed using a random effects model. Each rate was contrasted with placebo. Sensitivity analyses were performed to examine the robustness of the results.

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The purpose of this study was to use this method to investigate the Flagyl 400 Tablets effects of newer antidepressants on 5-HT(1A) receptor-mediated function.

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Venlafaxine may be beneficial to individuals with OCD, including those who have not responded to prior SSRI trials. However, these findings must be interpreted with caution, as the study is limited by its open, retrospective nature and its inclusion of patients with comorbid Crestor Generic Picture diagnoses and patients on concomitant medications. Prospective, controlled trials with a more homogeneous patient population are needed to replicate these preliminary findings.

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Treatment costs for 6 months were pound1530 for venlafaxine, pound1539 for fluoxetine and pound1558 for amitriptyline (year of costing 2006). Cost effectiveness as assessed by incremental cost per QALY ratio at 8 weeks was pound20 600 for venlafaxine compared with fluoxetine, with Prilosec Mg fluoxetine dominating (being less costly and more effective than) amitriptyline. To test the robustness of the model a Rank Order Stability Assessment was performed that showed that even if fluoxetine and/or amitriptyline were given away free, a scenario starting with venlafaxine would still be the least costly treatment over a 6-month period.

effexor with alcohol 2015-12-04

An 18-year-old woman ingested 22 g of venlafaxine with suicidal attempt. At admittance two hours after ingestion she had Tofranil Tablets 10mg serotonine syndrome, repeated seizures and hypotension. After eight hours malignant arrythmias culminated in cardiac arrest, which was successfully resuscitated. Progressive liver and cardiac failure lead to fatal outcome after 48 hours. It is discussed whether early lipid infusion should be given in case of ingestion of high doses of venlafaxine, before myoclonia, seizures, hypotension, rhabdomyolysis and liver failure developed, since these can only be treated symptomatically.